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The San Diego Fire Fighters & Paramedics

12 Lead E.C.G. Field Diagnosis Made Easy

rev. 09-03-97

Helpful Hints and Rules
Electrode Placement
12 Lead Quick Triage
Systematic Infarct Recognition Approach
12 lead rapid assessment
Rapid 12 Lead Interpretation
Bundle Branch Block
Infarct Recognition
Axis Deviation

Knowledge of the anatomy of the heart, the physiology of the heart, and the electrocardiography of the heart make a paramedic an
Emergency Cardiac Care Provider.

As primary point of care field paramedics, we have been taxed with the new challenge of diagnosing 12 lead cardiographs in the field and making a definative diagnosis of acute myocardial infarction. This overwhelming task has been at sometimes difficult to manage and to complicate matters, we do not use this skill regularly enough to maintain the skills.

So, I hope the following information is helpful...

The 12 lead EKG measures electrical potential

All cells membranes in the body are charged.
In a polarized state (resting) the membrane carries a net + charge. (results from the difference of intra and extra cellular electrolyte concentrations)

Depolarization: The shift in electrolytes which reverses the charge

Repolarization: Return to the resting state

Helpful hints and rules to reading the 12 lead...

Always be sure to check for an inverted or negative QRS complex in AVR. This will ensure that the lead were correctly connected appropriately and your recordings shold be accurate.
Analize and evaluate the right chest leads. V1 & V2 will reveal more than any other two contigeous leads. This is where you check for a Bundle Branch Block, Anterior and Posterior wall infarctions, and "R" wave progression, ect...
When checking your axis, always focus on Leads I & AVF. Be sure to check for Bundle Branch Block. Axis vectors are inaccurate in their pressence.
When checking for signs of infarct, omit AVR. It is of no diagnostic value due to the fact that it misrepresents pathological Q waves and obscures them.
Acute Myocardial Infarction cannot be positively identified in the pressence of LBBB!!!
It is prudent to suspect it per the patients presentation: however, serum enzyme tests among other things are needed to make the diagnosis.
As a rule, standard criteria for diagnosing AMI dictates that ST elevation of 1 mm. or more in the presence of pathological Q waves in 2 or more contigous leads is sufficient.

Three kinds of leads:

Standard Limb Leads
Augmented Lead
Precordial Leads

Electrode Placement:

Einthovens Triangle represents the leads that we all use with our monitors on a regular basis:

1) Standard Limb Leads

Lead I: The positive lead is above the left brest or on the left arm and the negative lead is on the right arm.
Records the difference of potential between the Left arm and Right arm.
Lead II: The postive lead is on the left abdomen or left thigh and the negative lead is also on the right arm.
Records the difference of potential between the left leg and the right arm.
Lead III: The postive lead is also on the left abdomen or left lower lateral leg but the negative lead is on the left arm.
Records the difference of potential between the left leg and the right arm.

The hexaxial view

Leads I, II & III & AVR, AVL, and AVF.

I: Left Chest
II: Left Upper Quadrant
III: Right Upper Quadrant
AVR: Right lateral arm
AVL: Left lateral arm
AVF: Right lateral lower leg

2) Augmented Leads

The four limb leads go on the four extremities as follows:

The upper extremities need placement of the electrodes on the area of the lateral humoral aspect of the arms.

The lower extremities need placement of the electrodes on the lateral lower legs near the lateral mallelous.
Lead aVR faces the heart from the right shoulder and is oriented to the cavity of the heart.
Lead aVL faces the heart from the left shoulder and is oriented to the Left Ventriacle.
Lead aVF face the heart from the left hip and is oriented to the inferior surface of the Left Ventricle.

3) Precordial Leads

Six Precordial Electrode Placement:

Records potential in the horizontal plane. Each lead is positive.

The major forces of depolariztion move from right to left.

V1 and V2 are negative deflections.

V3, V4, V5 and V6 become more positive ( peak positive is V3 or V4 ).

V1 - fourth intercostal, right strernal border.

V2 - fourth intercostal, left sternal border.

V3 - equal distance between V2 and V4.

V4 - fifth intercostal, left mid clavicular line.

V5 - anterior axillary line, same level with V4.

V6 - mid axillary line, same level with V4 and V5.

The precordial views make up a cross section view of the heart in a transverse horizontal plane projecting a view across the AV Node.

Einthovens Triangle and the four limb leads make up the "HEXAXIAL VIEW!" This view is a vertical/frontal-posterior - ventral/dorsal plane making a star with 6 points intersecting through the heart in a flat frontal plane across the patients chest.

The PRECORDIAL views are used to make up the other six veiws of the heart for a total of twelve views.

So, adding this up: lead I, II and III, lead AVR, AVL, AVF, and the 6 precordial leads equals 12 leads... RIGHT?????

Correct, however, we only need 10 electrodes palced.
( 4 on the limbs and 6 on the chest )
The cardiac monitor uses the four Limb Leads to make up Lead I, II, III & AVR, AVL, AVF; six views...

12 lead Quick Triage

The following situation consistutes activation of the cardiac response team at the hospital by reporting the field daignosis of AMI!

Category one: AMI that clearly meets the criteria.

Example:

1 mm or more of ST elevation in the inferior leads (II, III, AVF) with reciprocal changes in the lateral leads (I, AVL, V5, V6)

Reciprocal changes not necesssary to make the diagnosis.
Category two: The following will result in your reporting the specific findings of concern that may or may not result in the Cardiac Response team.

Example:

1 mm of ST elevation in the anterior leads. (V1-V4)

Example:

Injury/Infarct pattern in the presence of LBBB with cardiogenic clinical presentaion.

The following situation will result in the 12 Lead ECG being reported as "normal". No subsequent activation of the cardiac response team.

Category Three: No patterns of ischemia or infarction.

Other Signals to use as a diagnostic tool:

Tackycardia: (heart rate above 100) indicates damge to the left Ventricle and an "anterior" or "lateral" infarct. The Left Circumflex and or Left Decending Coronary Artery is occluded.
Visable elevation in the CHEST LEADS: V-3, 4, 5, & 6.
Bradycardia: (heart rate below 60) indiactes damage to the Right Ventricle and an "inferior" or "posterior" infarct. The Right Coronary Artery is occluded.
Elevation in the LIMB LEADS: II, III, & AFV.

Systematic Infarct Recognition Approach

Assure that aVR is priamarily negative.
Rule out a :eft Bundle Branch Block (LBBB) in V1 and or V2... Verify in V6.
Check all leads for patterns of ischemia, injury, infarction and reciprocal changes.

AMI diagnosis criteria: 1mm. or more of ST elevation in 2 or more contiguous leads.
Anterior wall reqires 2mm. or more of ST elevation (V1-V4)
Caution: LBBB

Lead Groups

INFARCT LOCATION:	ST ELEVATION FOUND IN:
Anterior - Septal	V1, V2, V3, and V4 -- 0.2mV or more in leads
Posterior	V1, and V2 -- 0.2mV or more in leads
Inferior	II, III, and aVF -- 0.1mV or more in 2 leads
High Lateral	I, and aVL -- 0.1mV or more in 2 leads
Low Lateral	V5, and V6 -- 0.1mV or more in 2 leads

"ST Depression indicates Angina"

Diagrams below indicate which part the heart is being affected and what lead would show the changes.

Reciprocal Changes

Region of ST Elevation	Region of ST Depression
Anterior (leads V1-V4)	Inferior (true posterior)
Inferior (leads II, III, aVF)	Anterior (leads V1-V3 or lateral lead 1. aVL)
Lateral ( leads I, aVF, V5, V6)	Inferior ( leads II, III, aVF)
True Posterior	Anterior (leads V1-V3)

12 lead rapid assessment

Verify aVR is negative
Assess rate and rhythm
Axis determination - Leads I and aVF
Conduction abnormalities:

LBBB - seen in V1
Hypertrophy
Aneurysm
Pericarditis
Drugs or Electrolytes
Early repolarization
Ischemia, Injury, Infarct signs:

T-wave inversions

ST segment elevation

Significant Q waves
Acute MI pattern:

Anterior:

ST elevation in V1, V2, V3, V4

ST depression in II, III, aVF

Inferior: ST elevation in II, III, aVF

ST depression in V1, V2, V3, or I, aVL

Lateral: ST elevation in I, aVL, V5, V6

ST depression in II, II, aVF

Septal wall: ST elevation in I, aVL, V1, V2
Posterior: tall and wide R waves and ST depression in V1, V2
Right Ventricular: ST elevations in V4R, V5R, V6R

(5 additional right chest wall electrodes placed on the chest in the same positions as the precordial leads)
Clinical pressentaion
Treatment plan

Electrical Current:

Electricity always flows from positive to negative. The electrical current should flow from negative to positive in the normal healthy heart. So, if this pattern is disrupted by a "detour" or as in the heart, "an infarct" or "injury" the ECG recording will indicate the abnormal flow of current. With an infarcted heart, the electrical current flows opposite of where it is expected to flow. Hence, the elevated or depressed ST segment . For instance, an inferior infaction will show an elevation in lead II, III and aVF. The normal flow would be "isoelectrical" and the ST segment would be equalized or level. But, since the flow is going backwards around the damaged heart muscle, we see an elevation on the record. It is this precise measurement that can dictate exactly where the infarct is located. If the ST segment is elevated in V2, V3 and V4, the infarct is anterior. These views are looking at the front or anterior area of the heart muscle. The current is flowing toward the positive electrode on the patients chest. When the current is disrupted, it will show as an elevation in the ST segment versus an isoelectric reading.

Think of it like this: An Xray film is placed behind the heart at the area between the Ventrical Septum. The X-Ray machine shoots the picture from the anterior heart directly above the film. and the film catures the image. We would be looking at the area of the heart at the Septal region which would be in ECG terminology V3.

The infract area will have no electrical current. The ST segment will be depressed
The injured area will have ST elevations and will release Cardiac Muscle enzimes. These enzimes are CK, MyoGlobin and Troponin I
The ishemic area may have ST elevations and ECG cahnges to include PVC's, PAC's and PJC's.

The 12 Lead Photograph

**Simulatneous aquisition 2.5 seconds per view, 10 seconds for a complete study**
I	aVR	V1	V4
II	aVL	V2	V5
III	aVF	V3	V6

12 lead ECG; a real time vedio recording of the hearts electrical function.

This record indicates a "septal / anterior Infarct."

If you can comprehend which way the current is expected to flow in The HEXAXIAL VIEW and The PRECORDIAL VIEW of the heart, then you can diagnose which area is effected if it is an abnormal flow...

See the information below.

12 lead rapid interpretation

**Common ECG Formation**
	Iscehmia=Inverted T waves Inverted T wave is symmetrical T waves are usually upright in leads I, II, and V2-V6
	Injury=Elevated ST segment Signifies an acute process; ST returns to baseline with time If ST elevation is diffuse and unassociated with Q wavesor reciprocal ST depression, consider pericarditis Location of injury can be determined in same manner as infarct location Usually associated with reciprocal ST depression in other leads
	Infarction=Q wave Small Qs may be normal in V5, V6, I and aVL Abnormal Q must be one small square (0.04 sec) wide Also abnormal if Q-wave depth is greater than one-third of QRS height in lead III

Making the accurate Field Diagnosis:

There are elevations ( 1 mm )in two contegous (connecting) leads:
Leads adjacent to eachother...
There is at least one lead with reciprocal changes..
If the Q wave is more than 1/3 the size of the R wave...

Table below shows what the ECG would look like in the Vector where the heart is being affescted. All other areas would look normal, without elevation or depression. unless there is an "old MI." In that case, the prior damage would show up as a depressed segment.

**Anterior Infarction**
	ST elevation without abnormal Q wave Usually associated with occlusion of the left anterior decending branch of the left coronary artery (LCA)

**Lateral Infarction**
	ST elevation with/without abnormal Q wave May be a component of a mutiple-site infarction Usually associated with abstruction of the left circumflex artery

**Inferior Infarction**
	ST elevation with/without abnormal Q wave Usally associated with right coronary artery (RCA) occlusion

**Right Ventricular Infarction**
	Usually accompanies inferior MI due to proximal acclusion of the RCA Best diagnosed by 1 - 2 mm ST elevation in lead V4R An important cause of hypotension in inferior MI recognized by jugular venous distension with clear lung fields Aggressive therapy is indicated, including: reprofussion, adequate IV fluids for right heart filling, and pacingf to maintain A-V synchrony if necessary

**Poterior Infarction**
	Tall, broad (>0.04 sec) R wavr and ST depression in V1 and V2 (reciprocal changes) Frequestly associated with inferior MI Usually associated with obstruction of RCA and or left circumflex coronary artery

Pathological Q waves:

If the Q wave ( the first downward "negative" deflected wave ) is more than 1/3 the size of the R wave ( the first upward deflected "positive" wave ) it is pathological and indicative of an A.M.I.

If no R wave is recorded, then the infarct is extreamly acute. There is no electrical activity of the ventricle durring polarization and contraction.

Bundle Branch Block In Bundle Branch Block, the firing of the Ventricles does not occur simultaneously as it should (It occurs in series instead of parallel). Conduction reaches a block in one of the branches (in the cardiac septum) and refers it to the opposing branch to be conducted completely. It is then when conduction jumps the Intra-Ventricular Septum to ultimately conduct to the remaining blocked Bundle Branch. It is because of this that you see two different distinctly separate QRS complexes over-lapping one another. Hence, the "Rabit Ear" and "RSR pattern." Remember, the QRS complex will always be at least .12 in width and posses abnormal morphology. ALWAYS CHECK RIGHT AND LEFT CHEST LEADS FOR BUNEDLE BRANCH BLOCK (V-1, V-2, & V-5, V-6) Infarction associated with a
Left Bundle Branch Block

A LBBB may result from an acute myocardial infarction (AMI), but field paramedics cannot dianose AMI in the presence of LBBB. The presence of LBBB negates meaning ful interpretation of other EKG criteria

A LBBB pattern prior to the onset of clinical findings of AMI with marked reduction in voltage of the QRS complex may offer clues to the diagnosis of an infarction.

LBBB obscures the pattern of AMI since the initial QRS vector is abnoemally directed in a LBBB pattern. It will obscure the infarction vector and abnormal Q waves will not appear. The most diagnostic feature of AMI is the abnormal direction of the initial 0.04 sec of the QRS vector (ie; the abnormal Q wave).

LBBB is usually associated with an Inferior wall AMI when an AMI is diagnosed.
LBBB is usually associated with hypertensive ishemia or primary myocardial disease.

Diagnosing the Bundle Branch Block:

Right or Left???

The last 0.04 seconds of deflection on the QRS complex is used to determine the direction of the block.

In V1 or MCL1, if the QRS duration is greater that 0.12 seconds (usually 0.14 - 0.20 seconds) and the last 0.04 second segment of the complex is pointing down (negative deflextion), the block is LEFT.

If the last 0.04 seconds of the QRS complex is pointing up and is positively deflected, the block is RIGHT.

Infarct Recognition

Some Additional Tips...

Certain easily identifiable ECG changes that are observed in the presence of cardiogenic chest pain, reveal some strong presumptive evidence toward the positive diagnosis of AMI. This pattern of changes is refered to as the "evolution of Myocardial Infarction."

It is offen suggested that the first observable evolutionary change is the ischemia we associate with T-wave inversion or ST segmnet depression. Then, onto what is refered as the hyperacute phase. In the hyperacute phase of the MI, (usually the first few minutes) the T-wave may simply increase in height, and/or the ST segment becomes elevated. The finale phase is the acute phase. In the acute phase, (usually the first hour or more) the ST segment elevation is accompanied by the developement of a pathological Q wave. This Q-wave comfirms the diagnosis of MI.

This evolution is not precise, however. Often times the T-wave may invert in the presence of ST segmnet elevation durring the end of the hyperacute phase. In any event... the mose critical observation should be the recognition of ST elevation in 2 or contiguos leads. This is most important to paramnedic in the pre-hospital phase because the developement of the Q-wave may take hours and could easily be missed in the field.

Eventually, the ST segment will return to its baseline and the T-wave resumes its normal position, leaving only the Q-wave as evidence that an infarction has occurred. Recent research and studies have produced 95% accuracy in field diagnosis by paramedics. Perhaps some reasons would include other indications for ST changes. They would include simple angina, drug effects, and electrolyte imbalance.

Axis Deviation:

Use Lead I, II, and aVF to diagnois Axis Deviation

Vectors and Axis

Vector: A quantity of electrical force that has a known magnitude and direction.
Axis: A hypothetical line which joins the poles of a lead which measure electrical force.
Mean Cardiac Vector: The avaerage of all the instananeous vectors. ( AKA mean electrical axis ).

The postion of the mean cardiac vector provides information about the electrical "position" of the heart, and is influenced by the relationship of the heart within the chest, as well as by the anatomy of the heart itself.

For pre-hospital purposes, the axis is either "normal" or "not normal."

Normal Deviation:

The QRS deflection is upright or positive in I and either aVF or Lead II.

A normal axis means the QRS axis falls between 30 and 90 degress in the chest. The heart is lying in an angle between these parameters.
Right Axis Deviation:

The QRS is downward or negatively deflected in I and positive in aVF or Lead II.

The heart is lying in an angle lower the 30 dgress in the chest.

Can be normal in young adults or "thin peolpe."

May be abnormal in peolpe who have a block in the posterior division of the left bundle.

Can imply delayed activation of the right ventricle ( as seen in RBBB ) or Right Ventricular enlargement.

Pathology: Right Ventricular enlargemnet and hypertrophy. C.O.P.D. Pulmonary Embolism, Congenital heart Disease, Inferior wall MI.
Left Axis Deviation:

The QRS is uright or postively deflection in I and negative in aVF or Lead II.

The heart is lying in an angle greater than 90 degress in the chest.

Can be normal in the presence of acites, abdominal tumors, pregnancy or obesity.

Abnormalities are due to Left Ventricular enlargement or a Left anterior hemiblock.

Pathology: Left ventricular enlargement, and hypertrophy, Hypertension, Aortic Stenosis. Ischemic Heart Disease. Inferior wall MI.

As stated above. the electrical current should flow to the positive lead. If it does not flow in a positive direction, the heart is pointing toward the upper right or the left. So, if the QRS is negative in aVF, the heart is pointing more to the left than noraml; hence, Left Axis Deviation. If the QRS is negative in Lead I, the heart is pointing more to the right than normal; hence, Right Axis Deviation.

This is very complicated and difiicult to explain in this forum. If you need info on AXIS deviation or 12 lead diagnosis, please send E-Mail and information will be provided by E-Mail or conventional postage.

By, Mitch Mendler E.M.T. paramedic, San Diego Paramedic.